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For instance (Molinari and Bromfield, 2023), demonstrated that while heat shock (40 °C for 24 h) of endometrial epithelial cells increases the gene expression of HSPA1A and amplified inflammatory responses to LPS (1 or 10 µg/mL), knock down of HSPA1A or heat shock factor 1 ( HSF1 ) exacerbated the immune response to LPS

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Antioxidant and anti-inflammatory substances, such as D-mannose, icaritin (162) and Fer-1, inhibit ferroptosis in chondrocytes by regulating the GPX4 pathway, thereby slowing cartilage degeneration and decelerating the pathological process of OA (163)