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this includes chlorambucil (adduct formation through nucleophilic substitution of the atom of chlorine [118]), cyclophosphamide (acrolein is one its main metabolites conjugating to GSH and becoming significantly less cytotoxic than the parent compound [119]), mitomycin C (forming mono- and bis-conjugates with GSH to be detoxified [120]), and various tyrosine kinases inhibitors (such as axitinib and sunitinib [121])