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Here we noted that U87 cells treatment with BiNPs-NISM-Dox/Pi3Ki@BSA and BiNPs-NISM-Dox/Pi3Ki@BSA-GSH-Glu nanosystems showed higher rate migration inhibition, represented the synergic dual action of Dox-induced DNA damage and Pi3K inhibition and also remarked the importance of targeting for elevation the treatment efficacy
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Sh1) were detected in the embryo in non-infected kernels, but the gene was up-regulated in the aleurone and scutellum under A
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