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The mechanisms of the GH–IGF-I axis in the regulation of normal human growth is discussed followed by descriptions of mutations in GHR , STAT5B , IGF-I , IGFALS , IGF1R , and GH1 defects causing bio-inactive GH or anti-GH antibodies

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This mechanism of action probably plays a major role, as inflammations are causally involved in the development of nerve damage in most neurological diseases